Nutrients | Free Full-Text | The Dietary Intake of Wheat and other Cereal Grains and Their Role in Inflammation | HTML

https://www.mdpi.com/2072-6643/5/3/771/htm


In this review we discuss evidence from in vitro, in vivo and human intervention studies that describe how the consumption of wheat, but also other cereal grains, can contribute to the manifestation of chronic inflammation and autoimmune diseases by increasing intestinal permeability and initiating a pro-inflammatory immune response


De Punder, K. & Pruimboom, L. (2013). The Dietary Intake of Wheat and other Cereal Grains and Their Role in Inflammation. Nutrients, 5(3). doi:10.3390/nu5030771

Natural forms of vitamin E: metabolism, antioxidant, and anti-inflammatory activities and their role in disease prevention and therapy. - PubMed - NCBI

http://www.ncbi.nlm.nih.gov/pubmed/24704972

Natural cocoa consumption: Potential to reduce atherogenic factors? - PubMed - NCBI

Natural cocoa consumption: Potential to reduce atherogenic factors? - PubMed - NCBI 

http://www.ncbi.nlm.nih.gov/pubmed/25769436




Natural cocoa consumption resulted in a significant decrease in haptoglobin (P=.034), EMP concentration (P=.017) and monocyte CD62L (P=.047) in obese compared to overweight and normal-weight subjects. Natural cocoa consumption regardless of BMI group was associated with an 18% increase in high-density lipoprotein (P=.020) and a 60% decrease in EMPs (P=.047). Also, obese subjects experienced a 21% decrease in haptoglobin (P=.034) and a 24% decrease in monocyte CD62L expression in (P=.047) following 4 weeks of natural cocoa consumption. Collectively, these findings indicate that acute natural cocoa consumption was associated with decreased obesity-related disease risk.

Food Additives Alter Gut Microbes, Cause Diseases in Mice - NIH Research Matters - National Institutes of Health (NIH)

Food Additives Alter Gut Microbes, Cause Diseases in Mice - NIH Research Matters - National Institutes of Health (NIH)

http://www.nih.gov/researchmatters/march2015/03162015additives.htm

At a Glance

• Common food additives called emulsifiers promoted colitis and metabolic syndrome in mice by altering gut microbes.

• The findings suggest that certain food additives might play a role in the increasing incidence of obesity and chronic inflammatory bowel disease.

The Best Diet For Weight Loss | David Perlmutter M.D.

http://www.drperlmutter.com/best-diet-weight-loss/

Diabetes and Risk for Cognitive Decline | David Perlmutter M.D.

http://www.drperlmutter.com/diabetes-risk-cognitive-decline/

Meta-analysis: Vitamin D supplementation reduces specific inflammatorymarker

http://www.vitamindcouncil.org/blog/meta-analysis-vitamin-d-supplementation-reduces-specific-inflammatory-marker/?mc_cid=3e1b321425&mc_eid=1d72be5c3b

Fatty acids, inflammation, and asthma. [J Allergy Clin Immunol. 2014] - PubMed - NCBI

Fatty acids, inflammation, and asthma. [J Allergy Clin Immunol. 2014] - PubMed - NCBI

De-Inflame with SuperfoodsDrJockers.com

De-Inflame with SuperfoodsDrJockers.com

Infections, Inflammation, and the Risk of Coronary Heart Disease

Infections, Inflammation, and the Risk of Coronary Heart Disease

carbohydrate diet has a favourable impact on low-grade inflammation in type 2 diabetes compared with advice to follow a low-fat diet

http://informahealthcare.com/doi/full/10.3109/07853890.2014.894286
or
http://informahealthcare.com/doi/pdf/10.3109/07853890.2014.894286 

Background. 

Infl ammation may play an important role in type 2 diabetes. It has been proposed that dietary strategies can modulate infl ammatory activity. Methods. We investigated the eff ects of diet on infl ammation in type 2 diabetes by comparing a traditional low-fat diet (LFD) with a low-carbohydrate diet (LCD). Patients with type 2 diabetes were randomized to follow either LFD aiming for 55 – 60 energy per cent (E%) from carbohydrates ( n 30) or LCD aiming for 20 E% from carbohydrates ( n 29). Plasma was collected at baseline and after 6 months. C-reactive protein (CRP), interleukin-1 receptor antagonist (IL-1Ra), IL-6, tumour necrosis factor receptor (TNFR) 1 and TNFR2 were determined.

Results. 

Both LFD and LCD led to similar reductions in body weight, while benefi cial eff ects on glycaemic control were observed in the LCD group only. After 6 months, the levels of IL-1Ra and IL-6 were
signifi cantly lower in the LCD group than in the LFD group, 978 (664 – 1385) versus 1216 (974 – 1822) pg/mL and 2.15 (1.65 – 4.27) versus 3.39 (2.25 – 4.79) pg/mL, both P 0.05.

Conclusions. 

To conclude, advice to follow LCD or LFD had similar eff ects on weight reduction while eff ects on infl ammation diff ered. Only LCD was found signifi cantly to improve the
subclinical infl ammatory state in type 2 diabetes.
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Key messages

• In type 2 diabetes, randomization to advice to follow a low-carbohydrate diet or a low-fat diet had
similar eff ects on weight reduction, while eff ects on infl ammation diff ered.
• Aft er 6 months, the low-carbohydrate diet, but not the low-fat diet, had a favourable impact on low-grade infl ammation in type 2 diabetes.