Supplementation with antioxidants may preclude these health-promoting effects of exercise in humans.
Svenska via Google translate: Tillskott med antioxidanter kan hindra dessa hälsofrämjande effekter av träning på människor.
Biological Sciences - Medical Sciences: Michael Ristow, Kim Zarse, Andreas Oberbach, Nora Klöting, Marc Birringer, Michael Kiehntopf, Michael Stumvoll, C. Ronald Kahn, and Matthias Blüher
Antioxidants prevent health-promoting effects of physical exercise in humans PNAS 2009 106 (21) 8665-8670; published ahead of print May 11, 2009, doi:10.1073/pnas.0903485106
Results:
...
Dietary cholesterol was not statistically significantly associated with any coronary artery disease...
ischemic stroke or hemorrhagic stroke.
...
Dietary cholesterol did not statistically significantly change serum triglycerides or very-low-density lipoprotein concentrations.
Dietary cholesterol and cardiovascular disease: a systematic review and meta-analysis.
Berger S, Raman G, Vishwanathan R, Jacques PF, Johnson EJ.
Am J Clin Nutr. 2015 Aug;102(2):276-94. doi: 10.3945/ajcn.114.100305. Epub 2015 Jun 24.
PMID: 26109578
The worldwide association of H. erectus with elephants is well documented and so is the preference of humans for fat as a source of energy. We show that rather than a matter of preference, H. erectus in the Levant was dependent on both elephants and fat for his survival. The disappearance of elephants from the Levant some 400 kyr ago coincides with the appearance of a new and innovative local cultural complex – the Levantine Acheulo-Yabrudian and, as is evident from teeth recently found in the Acheulo-Yabrudian 400-200 kyr site of Qesem Cave, the replacement of H. erectus by a new hominin. We employ a bio-energetic model to present a hypothesis that the disappearance of the elephants, which created a need to hunt an increased number of smaller and faster animals while maintaining an adequate fat content in the diet, was the evolutionary drive behind the emergence of the lighter, more agile, and cognitively capable hominins. Qesem Cave thus provides a rare opportunity to study the mechanisms that underlie the emergence of our post-erectus ancestors, the fat hunters.
Citation: Ben-Dor M, Gopher A, Hershkovitz I, Barkai R (2011)
Man the Fat Hunter: The Demise of Homo erectus and
the Emergence of a New Hominin Lineage in the Middle Pleistocene (ca. 400 kyr)
Levant. PLoS ONE 6(12): e28689. doi:10.1371/journal.pone.0028689
Ayyadurai, V.A.S. and Deonikar, P. (2015) Do GMOs
Accumulate Formaldehyde and Disrupt Molecular Systems Equilibria? Systems
Biology May Provide Answers. Agricultural Sciences, 6,
630-662. doi:10.4236/as.2015.67062.
there
is a bit of concern about the low salt diet. You can lower blood pressure okay
nobody's disputing that but there's a price to pay, you're going to raise a
renin and aldosterone levels fairly
substantially. So while you may lower your blood pressure you may actually put
yourself at higher risk have coronary artery disease.
there
was one group that it looked like urs actually very harmful to follow a
low-salt diet back group was the cardiac patients, so the people with heart
failure the ones I you all tell to eat a low salt diet, they seem to do much
worse, much worse than everybody else
25:30 - Potential
Harm From Too Little Salt Intake (The National Academies of Sciences)
Especially
for those people with heart failure you want to be very very careful because
the low salt diet might be extremely harmful to you okay it's not only not good
for you but it might be actually very harmful to you and that everything else
was that there was a really enough
Evidence
indicates that low sodium intake may lead to risk of adverse health effects
among those with mid- to late-stage heart failure who are receiving aggressive
treatment for their disease
The increased risk of being affected by a double diagnosis of type 1 diabetes and coeliac disease was modulated by perinatal risk factors. This suggests that early life events are important when it comes to children with type 1 diabetes also developing coeliac disease.
Leslie MA, Cohen DJA, Liddle DM, Robinson LE, Ma DWL. A review of the effect of omega-3 polyunsaturated fatty acids on blood triacylglycerol levels in normolipidemic and borderline hyperlipidemic individuals. Lipids in Health and Disease. 2015;14:53. doi:10.1186/s12944-015-0049-7.
Cameron E, Pauling L. Supplemental ascorbate in the supportive treatment of cancer: Prolongation of survival times in terminal human cancer. Proceedings of the National Academy of Sciences of the United States of America. 1976;73(10):3685-3689.
PMCID: PMC431183 http://www.ncbi.nlm.nih.gov/pmc/articles/PMC431183/
Cameron E, Pauling L. Supplemental ascorbate in the supportive treatment of cancer: Reevaluation of prolongation of survival times in terminal human cancer.Proceedings of the National Academy of Sciences of the United States of America. 1978;75(9):4538-4542.
PMCID: PMC336151 http://www.ncbi.nlm.nih.gov/pmc/articles/PMC336151/
Cabanillas F. Vitamin C and cancer: what can we conclude--1,609 patients and
33 years later? P R Health Sci J. 2010 Sep;29(3):215-7. Review. PubMed
PMID: 20799507. http://www.ncbi.nlm.nih.gov/pubmed/20799507
Recent meta-analyses have found no association between heart disease and dietary saturated fat; however, higher proportions of plasma saturated fatty acids (SFA) predict greater risk for developing type-2 diabetes and heart disease. These observations suggest a disconnect between dietary saturated fat and plasma SFA, but few controlled feeding studies have specifically examined how varying saturated fat intake across a broad range affects circulating SFA levels. Sixteen adults with metabolic syndrome (age 44.9±9.9 yr, BMI 37.9±6.3 kg/m2) were fed six 3-wk diets that progressively increased carbohydrate (from 47 to 346 g/day) with concomitant decreases in total and saturated fat. Despite a distinct increase in saturated fat intake from baseline to the low-carbohydrate diet (46 to 84 g/day), and then a gradual decrease in saturated fat to 32 g/day at the highest carbohydrate phase, there were no significant changes in the proportion of total SFA in any plasma lipid fractions. Whereas plasma saturated fat remained relatively stable, the proportion of palmitoleic acid in plasma triglyceride and cholesteryl ester was significantly and uniformly reduced as carbohydrate intake decreased, and then gradually increased as dietary carbohydrate was re-introduced. The results show that dietary and plasma saturated fat are not related, and that increasing dietary carbohydrate across a range of intakes promotes incremental increases in plasma palmitoleic acid, a biomarker consistently associated with adverse health outcomes.
Citation: Volk BM, Kunces LJ, Freidenreich DJ, Kupchak BR, Saenz C, Artistizabal JC, et al. (2014) Effects of Step-Wise Increases in Dietary Carbohydrate on Circulating Saturated Fatty Acids and Palmitoleic Acid in Adults with Metabolic Syndrome. PLoS ONE 9(11): e113605. doi:10.1371/journal.pone.0113605
EU-kommissionen har godkänt användningen av 17 genetiskt modifierade sorter av majs, soja, raps och bomull. De får användas till livsmedel och till foder. Godkännandet gäller import och bearbetning, men inte odling.
Produkterna tål vissa ogräsmedel eller insektsangrepp. Några av produkterna har bägge egenskaperna inbyggda. Flera av produkterna har ett tidigare godkännande, som nu har förlängts.
Majssorter
MON 87460,
NK 603
T25
Sojasorter
MON 87705,
MON 87769,
305423,
MON 87708,
GHB614XLLCotton25
BPS-CV127-9
Bomullsorter
MON 531xMON 1445,
MON 15985,
MON 88913,
T304-40,
MON 531,
MON 1445.
Rapssorter
MON88302
GT73.
Besluten sker i enlighet med Europaparlamentets och rådets förordning 1829/2003.
