Carbohydrate restriction as the default treatment for type 2 diabet... - PubMed - NCBI

http://www.ncbi.nlm.nih.gov/pubmed/18609058

Abstract

"Dietary carbohydrate restriction in the treatment of diabetes and metabolic syndrome is based on an underlying principle of control of insulin secretion and the theory that insulin resistance is a response to chronic hyperglycemia and hyperinsulinemia. As such, the theory is intuitive and has substantial experimental support. It has generally been opposed by health agencies because of concern that carbohydrate will be replaced by fat, particularly saturated fat, thereby increasing the risk of cardiovascular disease as dictated by the so-called diet-heart hypothesis. Here we summarize recent data showing that, in fact, substitution of fat for carbohydrate generally improves cardiovascular risk factors. Removing the barrier of concern about dietary fat makes carbohydrate restriction a reasonable, if not the preferred method for treating type 2 diabetes and metabolic syndrome. We emphasize the ability of low carbohydrate diets to improve glycemic control, hemoglobin A1C and to reduce medication. We review evidence that such diets are effective even in the absence of weight loss."

Carbohydrate restriction as the default treatment for type 2 diabetes and metabolic syndrome.

Feinman RD, Volek JS. Scand Cardiovasc J.
2008 Aug;42(4):256-63. doi: 10.1080/14017430802014838. Review.
PMID: 18609058

http://www.ncbi.nlm.nih.gov/pubmed/

Lisa Scherger - 'My Healthy Type 1 Son' - YouTube

https://www.youtube.com/watch?v=RtiduHZdbUg&feature=youtu.be

Lisa Scherger's blog: http://diabeticalien.blogspot.dk/

Weight Loss Recommendations Not in Line with Current Science | David Perlmutter M.D.

Weight Loss Recommendations Not in Line with Current Science | David Perlmutter M.D.

http://www.drperlmutter.com/weight-loss-recommendations-line-current-science/






Effects of Low-Carbohydrate and Low-Fat Diets
Ann Intern Med. 2014;161:309-318. doi:10.7326/M14-0180
http://www.drperlmutter.com/wp-content/uploads/2014/12/Effects-of-Low-Carbohydrate-and-Low-Fat-Diets.pdf

Conclusion:

The low-carbohydrate diet was more effective for weight loss and cardiovascular risk factor reduction than the low-fat diet. Restricting carbohydrate may be an option for persons seeking to lose weight and reduce cardiovascular risk factors.

Vitamin B1 och PMS Smärtor

Vid PMS besvär kan vitamin B1 vara ett bra alternativ. Det är en välbeprövat molekyl som inte har några biverkningar.

I en studie (1996) fick 556 personer 100 mg Thiamine hydrochloride, dagligen i 90 dagar och 87% av dessa kvinnor blev helt botade från menssmärtor, och 8 % hade lindrigare besvär än innan [1].

I en annan, nyare studie (2014) drar man slutsatsen:
"Det verkar som vitamin B1 är effektiv vid återvinning av psykiska och fysiska symptom på PMS. Därför kan detta vitamin användas för att nå ett viktigt mål för midwifery, det vill säga minskning av symtomens svårighetsgrad av PMS, utan några biverkningar." [2]

Kanske, kanske kan vitamin B1 också ha inverkan på humörstörningar? där finns iallafall en klar relation mellan just PMS och humörstörningar. [3]


[1] Curative treatment of primary (spasmodic) dysmenorrhoea.

Gokhale LB.

Indian J Med Res. 1996 Apr;103:227-31.

PMID: 8935744

http://www.ncbi.nlm.nih.gov/pubmed/8935744

[2] The effects of vitamin b1 on ameliorating the premenstrual syndrome symptoms.

Abdollahifard S, Rahmanian Koshkaki A, Moazamiyanfar R.

Glob J Health Sci. 2014 Jul 29;6(6):36220. doi: 10.5539/gjhs.v6n6p144.

PMID: 25363099

http://www.ncbi.nlm.nih.gov/pubmed/25363099

[3] Is There a Relationship between Mood Disorders and Dysmenorrhea?

Balık G, Ustüner I, Kağıtcı M, Sahin FK.

J Pediatr Adolesc Gynecol. 2014 Dec;27(6):371-4. doi: 10.1016/j.jpag.2014.01.108. Epub 2014 Sep 23.

PMID: 25256879

http://www.ncbi.nlm.nih.gov/pubmed/25256879

Killing fields: the true cost of Europe's cheap meat - The Ecologist

Investigations - Killing fields: the true cost of Europe's cheap meat - The Ecologist

http://www.theecologist.org/trial_investigations/336873/killing_fields_the_true_cost_of_europes_cheap_meat.html

GM soy and maize is toxic to rats – new detailed study

GM soy and maize is toxic to rats – new detailed study

http://gmwatch.org/index.php/news/archive/2014/15834

Ruminant for a healthier world: Märta Jansdotter at TEDxGöteborg - YouTube

Ruminant for a healthier world: Märta Jansdotter at TEDxGöteborg - YouTube

Sugar-Sweetened Beverages and Risk of Metabolic Syndrome and Type 2 Diabetes

http://care.diabetesjournals.org/content/33/11/2477.full

Consumption of artificially and sugar-sweetened beverages and incident type 2 diabetes in the Etude Epide´ miologique aupre` s des femmes de la Mutuelle Ge´ ne´rale de l’Education Nationale–European Prospective Investigation into Cancer and Nutrition cohort1 | David Perlmutter M.D.

http://www.drperlmutter.com/study/consumption-artificially-sugar-sweetened-beverages-incident-type-2-diabetes-etude-epide-miologique-aupre-s-des-femmes-de-la-mutuelle-ge-nerale-de-leducati/

Sunlight Holds Key To Killing Breast Cancer | GreenMedInfo | Blog Entry

Sunlight Holds Key To Killing Breast Cancer | GreenMedInfo | Blog Entry

http://www.greenmedinfo.com/blog/sunlight-holds-key-killing-breast-cancer-3

Dietary fats, carbohydrate, and progression of coronary atherosclerosis in postmenopausal women

Dietary fats, carbohydrate, and progression of coronary atherosclerosis in postmenopausal women

http://www.ncbi.nlm.nih.gov/pmc/articles/PMC1270002/

Mozaffarian D, Rimm
EB, Herrington DM. Dietary fats, carbohydrate, and progression of coronary
atherosclerosis in postmenopausal women. The American journal of clinical
nutrition 2004;80(5):1175-1184.

http://www.ncbi.nlm.nih.gov/pmc/articles/PMC1270002/

Abstract

Background: 
The influence of diet on atherosclerotic progression is not well established, particularly in postmenopausal women, in whom risk factors for progression may differ from those for men.

Objective: 
The objective was to investigate associations between dietary macronutrients and progression of coronary atherosclerosis among postmenopausal women.

Design: 
Quantitative coronary angiography was performed at baseline and after a mean follow-up of 3.1 y in 2243 coronary segments in 235 postmenopausal women with established coronary heart disease. Usual dietary intake was assessed at baseline.

Results: 
The mean (±SD) total fat intake was 25 ± 6% of energy. In multivariate analyses, a higher saturated fat intake was associated with a smaller decline in mean minimal coronary diameter (P = 0.001) and less progression of coronary stenosis (P =0.002) during follow-up. Compared with a 0.22-mm decline in the lowest quartile of intake, there was a 0.10-mm decline in the second quartile (P = 0.002), a 0.07-mm decline in the third quartile (P = 0.002), and no decline in the fourth quartile (P <0.001); P for trend =0.001. This inverse association was more pronounced among women with lower monounsaturated fat (P for interaction =0.04) and higher carbohydrate (P for interaction =0.004) intakes and possibly lower total fat intake (P for interaction =0.09). Carbohydrate intake was positively associated with atherosclerotic progression (P=0.001), particularly when the glycemic index was high. Polyunsaturated fat intake was positively associated with progression when replacing other fats (P = 0.04) but not when replacing carbohydrate or protein. Monoun-saturated and total fat intakes were not associated with progression.

Conclusions: 
In postmenopausal women with relatively low total fat intake, a greater saturated fat intake is associated with less progression of coronary atherosclerosis, whereas carbohydrate intake is associated with a greater progression.

Association of fructose consumption and components of metabolic syndrome in human studies: A systematic review and meta-analysis - Nutrition

Association of fructose consumption and components of metabolic syndrome in human studies: A systematic review and meta-analysis - Nutrition

Effect of low-calorie versus low-carbohydrate ketogenic diet in type 2 diabetes - Nutrition

http://www.nutritionjrnl.com/article/S0899-9007(12)00073-1/abstract

Physical Exercise as a Preventive or Disease-Modifying Treatment of Dementia and Brain Aging - Mayo Clinic Proceedings

Physical Exercise as a Preventive or Disease-Modifying Treatment of Dementia and Brain Aging - Mayo Clinic Proceedings





Mayo
Clinic Proceedings

September
2011 Volume 86, Issue 9, Pages 876–884

Physical
Exercise as a Preventive or Disease-Modifying Treatment of Dementia and Brain
Agin

http://www.mayoclinicproceedings.org/article/S0025-6196(11)65219-1/fulltext

Abstract:

A rapidly growing literature strongly suggests that exercise, specifically aerobic exercise, may attenuate cognitive impairment and reduce dementia risk. We used PubMed (keywords exercise and cognition) and manuscript bibliographies to examine the published evidence of a cognitive neuroprotective effect of exercise. Meta-analyses of prospective studies documented a significantly reduced risk of dementia associated with midlife exercise; similarly, midlife exercise significantly reduced later risks of mild cognitive impairment in several studies. Among patients with dementia or mild cognitive impairment, randomized controlled trials (RCTs) documented better cognitive scores after 6 to 12 months of exercise compared with sedentary controls. Meta-analyses of RCTs of aerobic exercise in healthy adults were also associated with significantly improved cognitive scores. One year of aerobic exercise in a large RCT of seniors was associated with significantly larger hippocampal volumes and better spatial memory; other RCTs in seniors documented attenuation of age-related gray matter volume loss with aerobic exercise. Cross-sectional studies similarly reported significantly larger hippocampal or gray matter volumes among physically fit seniors compared with unfit seniors. Brain cognitive networks studied with functional magnetic resonance imaging display improved connectivity after 6 to 12 months of exercise. Animal studies indicate that exercise facilitates neuroplasticity via a variety of biomechanisms, with improved learning outcomes. Induction of brain neurotrophic factors by exercise has been confirmed in multiple animal studies, with indirect evidence for this process in humans. Besides a brain neuroprotective effect, physical exercise may also attenuate cognitive decline via mitigation of cerebrovascular risk, including the contribution of small vessel disease to dementia. Exercise should not be overlooked as an important therapeutic strategy.